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| Journal of Medicinal Chemistry- February 14, 2013 Vol 56 No.3 |
About the cover page:
Novel AMF-26-induced Golgi disruption, apoptosis, and cancer-cell growth inhibition through a mechanism for preventing the ADP-ribosylation factor 1 activation. Golgi apparatus sorts and modifies hormones, enzymes, and other key proteins for transport elsewhere. Laboratory tests showed that the synthetic AMF-26 is just as effective as its natural counterpart and kills cancer cells differently than existing medicines.
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Total Synthesis of AMF-26, an Antitumor Agent for Inhibition of the Golgi System, Targeting ADP-Ribosylation Factor 1, isamu shiina et al., tokyo university of science.
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